PT  - JOURNAL ARTICLE
AU  - Douglas Lewis
AU  - Andrew Blow
AU  - Jonathan Tye
AU  - Tamara Hew-Butler
TI  - Considering exercise-associated hyponatraemia as a continuum
AID  - 10.1136/bcr-2017-222916
DP  - 2018 Mar 09
TA  - BMJ Case Reports
PG  - bcr-2017-222916
VI  - 2018
4099  - http://casereports.bmj.com/content/2018/bcr-2017-222916.short
4100  - http://casereports.bmj.com/content/2018/bcr-2017-222916.full
AB  - Exercise-associated hyponatraemia (EAH) always involves a component of overhydration relative to available exchangeable sodium stores. In the majority of cases, this is purely due to excessive consumption of fluids during exercise. In a lesser number of cases, it is apparent that excessive sodium loss through sweat may play a role by decreasing the amount of acutely available exchangeable sodium. Two cases demonstrating the latter, one in an individual with cystic fibrosis (CF) and another in an endurance athlete without CF, demonstrate how elevated dermal sweat losses may contribute to a relative dilutional EAH along a pathophysiological continuum.