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Posterior heel pain is a constellation of pathologies that may include retrocalcaneal bursitis, insertional tendonitis, paratenonitis and tendonosis among others.1 The diagnosis of insertional Achilles tendonitis can be established clinically and radiographically and must be differentiated from retrocalcaneal bursitis and Haglund's deformity.2 Patients present clinically with chronic pain of gradual onset typically relieved when the patient is unshod and it may be painful to perform a single leg raise.3 ,4 Calcaneal exostoses of the Achilles tendon may be evident on radiograph. Exostoses composed of calcium carbonate (CaCO3) are exposed to the more chemically active acetate radical and replace the carbonate forming the blood soluble calcium acetate molecule (Ca(C3H3O2)2).5
A treatment algorithm has been developed for posterior heel pain.3 Initial therapeutic modalities include physical therapy, orthotic management and anti-inflammatory medications.3 Iontophoresis has been suggested as an alternative to local corticosteroid injections.4 In a prospective study by Japour et al transdermal application of acetic acid through iontophoresis was shown to be an effective conservative treatment modality for heel pain.5
A 56-year-old man presented to a local podiatry clinic with intermittent posterior heel pain of several years duration that impeded the patients’ activities of daily living: driving, prolonged walking and golfing. Gait analysis of the patient revealed an antalgic gait with early heel rise on the left with a short step length of the right. The patient required one-hand assist on ascent and descent of stairs. The patient described the pain as achy, with the left heel more painful than the right. Radiographs revealed the presence of retrocalcaneal exostoses (figure 1).
The patient had objective improvement in measures such as range of motion and gait deviations from manual therapy techniques such as kinesiotaping, myofascial release, deep tissue massage, joint mobilisation, ultrasound (20% at 3.3 mHz, 2.0 W/cm2) and home exercises/self-care consisting of dorsiflexion stretching, and ice massage, however the pain was still present.
The patient was then treated with iontophoresis therapy of 4% acetic acid using phoresor PM900 at a setting of 2.0 mA DC current for 20 min over five times in 2 weeks in which the positive electrode was overlying the skin of the midshaft of the fibula, and the negative electrode centred over the insertion of the Achilles tendon bilaterally (figure 2). The patient completed the Foot and Ankle Ability Measure, Activities of Daily Living Subscale, and the Lower Extremity Functional Scale, prior to and after the iontophoresis therapy (figures 3 and 4). After introduction of the iontophoresis therapy, the patient's pain scale improved. Following the third iontophoresis treatment, the patient no longer needed the rail during stair ascent and decent, and after the fifth therapy the patient was discharged from physical therapy, reaching his goal of decreased posterior heel pain.
Iontophoresis can be utilised as an alternative to local corticosteroid injection for the treatment of posterior heel pain along with other conservative measures such as stretching, myofascial release and taping.
The multifactorial nature of posterior heel pain can be attributed to increasing age, weight gain, biomechanical faults, medications and a myriad of systemic disorders.2
Treatment algorithms suggest that after conservative treatment has been exhausted surgical intervention may consist of resection of the offending calcaneal spur and tendon debridement.
Competing interests None.
Patient consent Obtained.
Provenance and peer review Not commissioned; externally peer reviewed.
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