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A young alcoholic male patient was referred from another hospital with a history of loose stools and vomitings. There was documentation of hyponatraemia (Na-111 mEq/L) which was treated with intravenous normal saline. Next day, sodium was documented to be 134 mEq/L. On the third day, the patient had worsening of sensorium and was referred to our department for further management.
On presentation, the patient was comatose and had sluggishly reactive pupillary reflexes.
His MRI (figure 1) showed symmetrical hyperintensity in bilateral caudate and putamen …
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