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Eagle syndrome and carotid artery dissection: a rare skull base cause of stroke
  1. Shaun Selvadurai1,
  2. Andrew Williamson2,
  3. Jagdeep S Virk1,2 and
  4. Peter Clarke2
  1. 1Otolaryngology, Barts Health NHS Trust, London, UK
  2. 2Otolaryngology, Charing Cross Hospital, London, UK
  1. Correspondence to Shaun Selvadurai; shaun.selvadurai{at}nhs.net

Abstract

Eagle’s syndrome is a rare collection of symptoms that occur secondary to an elongated styloid process or calcified stylohyoid ligament irritating its surrounding structures. Classically, this presents as unilateral throat pain or rarely, as acute neurological symptoms secondary to compression of the internal carotid artery: so called ‘stylocarotid syndrome’. Significant neurological events in teenagers, secondary to Eagle syndrome have not been reported. We discuss the rare case of a teenage boy, diagnosed with right internal carotid artery dissection and middle cerebral artery infarction, with no cause initially identified. Following further admission with a transient neurological episode, he was noted to have elongated styloid processes with the right abutting the site of carotid dissection. He underwent styloidectomy and has since remained symptom free. This case highlights the importance of considering anatomical variants when assessing young patients with neurological symptoms, and the potential morbidity and mortality benefit that early surgical intervention may have.

  • neurological injury
  • stroke
  • otolaryngology / ENT
  • head and neck surgery

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Footnotes

  • Contributors SS edited and assisted drafting manuscript. AW edited and assisted drafting manuscript. JV, senior author, operated on patient, reviewed and edited manuscript. PC, senior author, operated on patient, reviewed and edited manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Case reports provide a valuable learning resource for the scientific community and can indicate areas of interest for future research. They should not be used in isolation to guide treatment choices or public health policy.

  • Competing interests None declared.

  • Provenance and peer review Not commissioned; externally peer reviewed.

  • Supplemental material This content has been supplied by the author(s). It has not been vetted by BMJ Publishing Group Limited (BMJ) and may not have been peer-reviewed. Any opinions or recommendations discussed are solely those of the author(s) and are not endorsed by BMJ. BMJ disclaims all liability and responsibility arising from any reliance placed on the content. Where the content includes any translated material, BMJ does not warrant the accuracy and reliability of the translations (including but not limited to local regulations, clinical guidelines, terminology, drug names and drug dosages), and is not responsible for any error and/or omissions arising from translation and adaptation or otherwise.

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