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Gastric dysplasia causing gastric outlet obstruction
  1. Rahul Kumar1,
  2. Ipsit Ilahi1,
  3. Tripti Prajapati2 and
  4. Pankaj Kumar Garg3
  1. 1Department of Surgical Oncology, All India Institute of Medical Sciences, Rishikesh, Uttarakhand, India
  2. 2Department of Radiodiagnosis, All India Institute of Medical Sciences, Rishikesh, Uttarakhand, India
  3. 3Department of Surgical Oncology, Shri Guru Ram Rai Institute of Medical and Health Sciences, Dehradun, Uttarakhand, India
  1. Correspondence to Dr Pankaj Kumar Garg; dr.pankajgarg{at}gmail.com

Abstract

Gastric dysplasia signifies the presence of atypical cells in the gastric mucosa, which have not invaded beyond the lamina propria, and it rarely leads to tissue growth large enough to cause gastric outlet obstruction (GOO) to the gastric contents. However, GOO is commonly observed as a first clinical manifestation of advanced invasive gastric cancer in developing countries where patients seek medical care late. The present case highlights the treatment journey of a young woman who presented to us with features of GOO. Her endoscopic and radiological findings revealed a nodular gastric antral thickening causing GOO. An endoscopic biopsy showed features of dysplasia. She underwent distal gastrectomy following discussion in a multidisciplinary tumour board. Histopathological examination of the gastrectomy specimen confirmed dysplasia without any invasion beyond lamina. To the best of our knowledge, this is perhaps the first report of dysplasia of the stomach presenting as GOO.

  • gastric cancer
  • gastrointestinal surgery
  • general surgery

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Footnotes

  • Twitter @DrPankajGarg

  • Contributors PKG—conceptualisation, patient care, writing the original draft and approval of final draft. RK—literature search, patient care, revision and approval of the final manuscript. TP—literature search, patient care, revision and approval of the final manuscript. II—literature search, patient care, revision and approval of the final manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Provenance and peer review Not commissioned; externally peer reviewed.

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