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Glucocorticoid-induced cardiomyopathy: unexpected conclusion
  1. Taha Sheikh1,
  2. Hina Shuja2,
  3. Syeda Ramsha Zaidi3 and
  4. Ayema Haque4
  1. 1Department of Internal Medicine, University of Toledo, Toledo, Ohio, USA
  2. 2Department of Internal Medicine, Karachi Medical and Dental College, Karachi, Pakistan
  3. 3Department of Internal Medicine, St Joseph Mercy Health System, Livonia, Michigan, USA
  4. 4Department of Internal Medicine, Dow University of Health Sciences, Karachi, Pakistan
  1. Correspondence to Dr Taha Sheikh; taha.sheikh{at}utoledo.edu

Abstract

Glucocorticoid excess is an under-recognised cause of cardiovascular adverse effects. The sources can be either endogenous (Cushing’s syndrome) or exogenous (Anabolic steroid abuse). Cardiovascular complications due to excess glucocorticoid includes hypertension, left ventricular hypertrophy, myocardial infarction, and heart failure. Although anabolic steroid-induced cardiomyopathy is a well-recognised phenomenon, endogenous corticosteroid-induced cardiomyopathy and heart failure are rarely reported sequelae of glucocorticoid excess in the body. We report a glucocorticoid-induced dilated cardiomyopathy in a 26-year-old African–American man with cushingoid features and symptomatic heart failure.

  • heart failure
  • adrenal disorders
  • drugs: endocrine system

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Footnotes

  • Contributors TS and SRZ were involved in the conception and design of the work. TS, AH and HS were involved in manuscript writing. TS and SRZ were involved in reviewing and revising the manuscript.

  • Funding The authors have not declared a specific grant for this research from any funding agency in the public, commercial or not-for-profit sectors.

  • Competing interests None declared.

  • Patient consent for publication Obtained.

  • Provenance and peer review Not commissioned; externally peer reviewed.