Toxic epidermal necrolysis: A review
References (40)
Acute disseminated epidermal necrosis types l, 2, and 3: study of sixty cases
J Am Acad Dermatol
(1985)- et al.
Management of Stevens-Johnson syndrome and toxic epidermal necrolysis in children
J Pediatr
(1989) - et al.
Toxic epidermal necrolysis from griseofulvin
J Am Acad Dermatol
(1988) - et al.
Fatal toxic epidermal necrolysis after griseofulvin
Lancet
(1989) - et al.
Toxic epidermal necrolysis
J Emerg Med
(1989) - et al.
Allergic cutaneous reactions to drugs
Prim Care
(1989) - et al.
Toxic epidermal necrolysis: an approach to management using cryopreserved allograft skin
J Am Acad Dermatol
(1987) - et al.
Mangement of toxic epidermal necrolysis in a pedatric burn center
Am J Dis Child
(1985) - et al.
Toxic epidermal necrolysis (Lyell syndrome)
Arch Dermatol
(1990) - et al.
Contact dermatitis; drug eruptions
Toxic epidermal necrolysis: an eruption resembling scalding of the skin
Br J Dermatol
Plasmapheresis in severe drug-induced toxic epidermal necrolysis
Arch Dermatol
Hyperbaric oxygen treatment of toxic epidermal necrolysis
Cutis
A burn center experience with toxic epidermal necrolysis
J Burn Care Rehabit
Improved burn center survival of patients with toxic epidermal necrolysis managed without corticosteroids
Ann Surg
Toxic epidermal necrolysis: clinical findings and prognosis factors in 87 patients
Arch Dermatol
Toxic epidermal necrolysis: a step forward in treatment
JAMA
Rehabilitative considerations for patients with severe Stevens-Johnson syndrome or toxic epidermal necrolysis: a case report
J Burn Care Rehabil
A review of TEN in Britain
Br J Dermatol
Corticosteroids for erythema multiforme?
Pediatr Dermatol
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Exfoliative diseases of the integument and soft tissue necrotizing infections
2018, Total Burn Care: Fifth EditionExfoliative Diseases of the Integument and Soft Tissue Necrotizing Infections
2017, Total Burn Care, Fifth EditionToxic epidermal necrolysis: Part I. Introduction, history, classification, clinical features, systemic manifestations, etiology, and immunopathogenesis
2013, Journal of the American Academy of DermatologyCitation Excerpt :As previously mentioned, there are a few exceptions in which “TEN without spots” exhibit >10% epidermal detachment on a large patch of erythema without preceding confluent purpuric macules or atypical flat target lesions. They most often begin with a prodrome of fever, malaise, anorexia, pharyngitis, headache, and rash, which may be morbilliform, defined as a fine, discrete maculopapular exanthem, or consist of atypical targetoid macules.16,32-35 Flaccid bullae, skin erosions, and painful inflammation and ulceration in the oral cavity comprise the major signs, which develop over a period of 1 day to 2 weeks33 (Fig 6).
Toxic epidermal necrolysis: Part II. Prognosis, sequelae, diagnosis, differential diagnosis, prevention, and treatment
2013, Journal of the American Academy of DermatologyCitation Excerpt :−11.5 + (0.1 × patient’s age + 0.03 × total BSA involvement + 5.75 if sepsis is present). Patients surviving the acute phase of TEN are also at risk for a host of sequelae (Table II), ranging from skin scarring and eruptive melanocytic nevi to vulvovaginal stenosis and dyspareunia.13-19 Ocular lesions are the most common complication of TEN, described in 20% to 79% of patients,14,20-22 and include dry eye syndrome, photophobia, a sandy sensation in the eye, symblepharon, corneal scarring, corneal neovascularization, corneal xerosis, trichiasis, reduced visual acuity, blindness, and subconjunctival fibrosis (Fig 2).3,13,23-25
Exfoliative diseases of the integument and soft tissue necrotizing infections
2012, Total Burn Care: Fourth EditionToxic epidermal necrolysis (Lyell's disease)
2010, Burns