Summary

Secondary glaucoma in thyroid eye disease (TED) is attributed either to raised episcleral venous pressure or to mechanical compression induced trabecular meshwork damage by mucopolysaccharide deposition. While the mechanism for raised intraocular pressure (IOP) in TED is largely mechanical and is often evident during its active stage, our case presented with aqueous misdirection in the left eye during the inactive stage. Appropriate diagnostic tests should be ordered to diagnose cause of raised IOP, which would help determine appropriate treatment for the cause for secondary glaucoma in TED.